Thanks, Jessica. Now we know why Luc Montagnier said that all the jabbed victims should be tested for AIDS. And as to why this virus was created, the billionaire psychopaths think we need to vastly reduce the population, and big Pharma wants to make hundreds of billions and doesn't care how many they have to murder to do it, and most politicians are narcissistic control freaks who have used fear to take away our rights.
So the injections might be a mass experiment of rolling the dice to see if injection induced spike might phenotype mix with HIV and create turbo HIV that might even be contagious like sars-cov-2? That would explain the paranoia in China about zero covid. I seem to recall that earlier this year a team of Chinese scientists also published on sars-cov-2 infecting CD4+ T-cell via something called LFA-1.
On another note, what the Boston U research makes me think of is the bivalent boosters. If the omicron spike is responsible for increased infectiousness of omicron and if pairing it with wuhan backbone makes it more deadly, is it really a good idea to start pumping out omicron spike in the bodies of injectees?
All these bad things bumping into each other seems...well....bad.
Oct 21, 2022·edited Oct 21, 2022Liked by Jessica Rose
Question #4
Could this enhanced virus be readily transmittable to others via the SARS route (nasal/air)?
Answer - yes, "it" can probably be transmitted to others via SARS.
But "it" can probably not be lethal via that route.
They've been trying this crap for decades, to make a bio weapon deadly enough to infect as aerosol and have not succeeded - unless INJECTED. Not saying we can't get sick that way, but using a nasal rinse early and often, no.
We certainly can be, and are being systemically weakened through fear, f****ing with food, air and water - basic biological needs of nutrients (minerals), sunshine and sleep.
So the hardest thing for me and perhaps all of my species at this time is to not let the bastards wear us down!
(1) the Gp120 epitopes on SC2 are not HIV-1 (so should not give cross reactive antibodies). When cross reactive antibodies occurred in the Australian vaccine trial that was because of another epitope used as a molecular clamp (to hold the trimer in place)
(2) The Gp120 epitope was added deliberately. This is all explained in this article and why they chose the Gp120
(3) Antibodies to Gp120 are enhancing. In other words, the more antibodies you create to this spike, the more infection you will see. They knew this and it totally explains why they were only ever after more and more *antibodies* instead of chasing *immunity*
(4) They developed an inhibitor but we were not allowed access to it. I published this on Gab as probably the only safe repository, and this gab includes a link to Doorless Carp's review too
Jessica, Zhengli Shi and cohorts proudly answered your question whether an "insert" from HIV was added to the Covid S-protein when they published that THEY DID EXACTLY THAT, in Journal of Virology, Feb. 2008, p. 1899–1907; Vol. 82, No. 4. From the article (emphasis is mine):
“In this study, we investigated the receptor usage of the SL-CoV S by combining a HUMAN IMMUNODEFICIENCY VIRUS-BASED PSEUDOVIRUS SYSTEM with cell lines expressing the ACE2 molecules of human, civet, or horseshoe bat ... In addition to full-length S of SL-CoV and SARS-CoV, a series of S chimeras was constructed by inserting different sequences of the SARS-CoV S into the SL-CoV S backbone ... a minimal insert region (amino acids 310 to 518) was found to be sufficient to convert the SL-CoV S from non-ACE2 binding to human ACE2 binding...”
Of course after working to perfect the human pathogenicity of this monster, Shi later published more of her progress, along with Ralph Baric and 13 other researchers (only one of whom was Chinese) in 2015. This was in Nature Medicine, where again they mentioned using an "HIV-based pseudovirus" and again, the emphasis is mine:
"...DBT cells (Baric laboratory, source unknown) expressing ACE2 orthologs have been previously described for both human and civet; bat Ace2 sequence was based on that from Rhinolophus leschenaulti, and DBT cells expressing bat Ace2 were established as described previously(8). Pseudotyping experiments were similar to those USING AN HIV-BASED PSEUDOVIRUS, prepared as previously described(10), and examined on HeLa cells (Wuhan Institute of Virology) that expressed ACE2 orthologs...."
But don't worry, all this was nothing to be concerned about, because the Nature Medicine article also states: "Synthetic construction of chimeric mutant and full-length SHC014-CoV was approved by the University of North Carolina Institutional Biosafety Committee and the Dual Use Research of Concern committee..." See? Uncle Ralph, said it was okay!
And there you have a YES to your other question, Jessica -- were recombinant techniques used at the Wuhan Institute of Virology to create something incredibly similar to SARS Cov-2, or possibly SARS Cov-2 itself? But did we even need that proof? Cuz... Sars Cov-2 was either engineered, or it naturally come from bats who inserted a Moderna-patented furin cleavage sequence into a virus while in their cave, then flew ~900 miles to Wuhan to spread it, where Chinese and American virologists had been working for years to turn a non-humanly-infectious horseshoe bat coronavirus into a lethal pathogen. And amazingly, those bats did so without infecting anyone along the way...
Here's the doi for the Journal of Virology article: doi:10.1128/JVI.01085-07. And for the Nature Medicine article: doi:10.1038/nm.3985.
Would love your take on those articles, Dr. Rose. And if you would, please use some recombinant techniques yourself, to create duplicates of your phenotype. We could really use a few more admirably relentless, Flamenco-dancing computational biologists who surf, to guide us through the science which is otherwise incomprehensible to us mortals. Thank you, thank you, for having the courage to share your brilliance with the rest of us.
The pandemic has been an odd blessing triggering dynamic alliances among like minded from every discipline & background on emergency footing crowdsourcing research and brainstorming solutions.
Can't wait for Nov 8th.. right up w Simon & Garfunkel in Central Park atm.. flippin adore you all!! tyvm
bioRxiv is where I first read about the furin cleavage site in a paper by Yuri Deigin (https://medium.com/@yurideigin/lab-made-cov2-genealogy-through-the-lens-of-gain-of-function-research-f96dd7413748)....this was shortly after a young 36-year-old doctor died from thrombocytopenia here in Miami....I was highly suspicious of the global shenanigans before that, but wow, after reading Deighin, I pulled out the tin foil hat...and then I found Team Enigma, then you, and McCullough, La Quinta Columna, and onward...thank you, Jessica. You are on the right side!
I remember well august 2022 with Ana Maria and Karen on Stew Peter Show, testifying that they had found HIV in the injections among fragmental dnas from insects and snakes, graphene and so on. For me I tried to get people´s attention to this, here on Substack, but to no avail until the whole dna fragment scandal was rather recently rolled out. Noone could imagine what has then become common knowledge, thousands of vaxxed deaths and more still coming on within this genocide.
Many years ago (about 1988) I was shooting a game of 8-ball with a med student who had just completed a clinical rotation through urology and had begun his rotation through psychiatry. The med student's urology professor was an old guy, who had done his urology residency at NYC's Bellevue Hospital in the late 1950s. One year a strange epidemic appeared in NYC's Haitian immigrant community, that seemed to be sexually transmitted and resulted in catastrophic failure of the immune system. Nearly all the 30-odd patients admitted with the condition, already had caught at least one secondary infection. All of them had skin lesions resembling Kaposi's Sarcoma, said the urology professor. None of them recovered. Most died within 30 days of hospital admission.
1) Was there a wild-type ancestor of the HIV virus running loose in Haiti in the 1950's?
2) Did it have a natural reservoir in some species found in Haiti?
3) Was HIV a less-lethal variant of the wild-type virus, that left enough living patients around to be studied?
I have zero knowledge beyond that, to contribute. What injected this disturbing news into our game of eight-ball, was my chance comment about interning in organic chemistry at what's now the Karmanos Cancer Institute in Detroit, for Dr Jerome Hurwitz, who instinctively kept closets and fridges full of samples of every hypothetical chemo drug his research group had ever made. Some decades before, one of the Karmanos Institute staff dreamt up a mechanism similar to the yet-undiscovered replication mechanism of retroviruses and postulated there might be cancers that reproduce in that way. After some discussion, they dreamed up the structure of Azidothymidine (AZT), got an NCI grant to study it, made a few dozen grams of it, and tested it in vitro on a number of types of cancer tissue to see if it would block them from growing. Result: Dismal failure. Not one cancer to date, reproduces like a retrovirus does.
Decades later, old Horwitz got a phone call from NIH asking if he had any hints for them on how to make AZT. Upon learning they were thinking it might solve the HIV epidemic, he shared his sample and some old notebook pages. At the time, a public health priority was to persuade at-risk patients to get tested for HIV. The mere possibility of a treatment could incentivize people to learn if they were infected and stop risky behaviors that could spread the infection.
I sliced my way to this answer with Occam's razor in one hand and Hanlon's in my other:
what better way to get rid of a germaphobe president? It was also, a dual-pronged attack (a tactic favorited by leftist operatives): Should he win? Two years in, EVERY vaccine injury is blamed on him. OR alternatively, it helps him lose.
Hi Jessica, a colleague just wrote this, your the first person I thought of, one b/c this is your knowledge wheel house and you love to investigate. Have you heard about this or did I just miss you writing on it? '“[O]n Wednesday, four months after vaccinations began in young children, Moderna investigators published [a] paper in the New England Journal of Medicine… Deep in the paper’s appendix, Moderna disclosed a case of new-onset Type 1 diabetes in a one-year-old girl that its investigators found was vaccine-related. This is a very serious vaccine-caused side effect that occurred in the trial and as far as I can tell has NEVER been mentioned anywhere else.
On page 62 of the paper’s 68-page appendix, in Table S26, Moderna charts all the side effects reported in the trial, serious or not. The footnotes to the Table S26 contain this note:
“the other SAE [serious adverse event] considered related was new-onset Type 1 diabetes mellitus and diabetic ketoacidosis in a 1-year-old female reported 37 days post dose 2.”
Based on the description in the footnotes, this incident was very dangerous. Diabetic ketoacidosis, or DKA, occurs when the body can no longer rely on its sugars for fuel because of a lack of insulin. Instead, it breaks down fat in an effort to survive, causing acids called ketones to build in the blood. DKA can be fatal if it is not quickly medically managed.
Neither the Centers for Disease Control report nor the Canadian report … mentioned this case; it is not clear why. The Moderna investigators also did not mention the case in the paper itself, only the appendix.
Type 1 diabetes occurs when body’s immune system attacks the pancreas and causes it to stop producing insulin. It is most frequently diagnosed in children and teenagers. It can occur and be diagnosed in infants and toddlers, but such diagnoses are very rare. A 1998 British paper found the annual incidence to be about 1 in 15,000 children.
Case reports from Japan and elsewhere have also linked the mRNA shots to the development of Type 1 diabetes in adults.” Alex Berenson, 10/21/22'
Thanks, Jessica. Now we know why Luc Montagnier said that all the jabbed victims should be tested for AIDS. And as to why this virus was created, the billionaire psychopaths think we need to vastly reduce the population, and big Pharma wants to make hundreds of billions and doesn't care how many they have to murder to do it, and most politicians are narcissistic control freaks who have used fear to take away our rights.
So the injections might be a mass experiment of rolling the dice to see if injection induced spike might phenotype mix with HIV and create turbo HIV that might even be contagious like sars-cov-2? That would explain the paranoia in China about zero covid. I seem to recall that earlier this year a team of Chinese scientists also published on sars-cov-2 infecting CD4+ T-cell via something called LFA-1.
On another note, what the Boston U research makes me think of is the bivalent boosters. If the omicron spike is responsible for increased infectiousness of omicron and if pairing it with wuhan backbone makes it more deadly, is it really a good idea to start pumping out omicron spike in the bodies of injectees?
All these bad things bumping into each other seems...well....bad.
Question #4
Could this enhanced virus be readily transmittable to others via the SARS route (nasal/air)?
Answer - yes, "it" can probably be transmitted to others via SARS.
But "it" can probably not be lethal via that route.
They've been trying this crap for decades, to make a bio weapon deadly enough to infect as aerosol and have not succeeded - unless INJECTED. Not saying we can't get sick that way, but using a nasal rinse early and often, no.
We certainly can be, and are being systemically weakened through fear, f****ing with food, air and water - basic biological needs of nutrients (minerals), sunshine and sleep.
So the hardest thing for me and perhaps all of my species at this time is to not let the bastards wear us down!
Hmmm. Matthew, Mark, Luc, and John and JJ, almost biblical
For sure. Important things to note:
(1) the Gp120 epitopes on SC2 are not HIV-1 (so should not give cross reactive antibodies). When cross reactive antibodies occurred in the Australian vaccine trial that was because of another epitope used as a molecular clamp (to hold the trimer in place)
(2) The Gp120 epitope was added deliberately. This is all explained in this article and why they chose the Gp120
https://arkmedic.substack.com/p/absolute-proof-the-gp-120-sequences
(3) Antibodies to Gp120 are enhancing. In other words, the more antibodies you create to this spike, the more infection you will see. They knew this and it totally explains why they were only ever after more and more *antibodies* instead of chasing *immunity*
see:
https://t.me/arkmedic/5593
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC135957/
https://pubmed.ncbi.nlm.nih.gov/25911103/
(h/t/ Jo Deinert for that paper)
(4) They developed an inhibitor but we were not allowed access to it. I published this on Gab as probably the only safe repository, and this gab includes a link to Doorless Carp's review too
https://gab.com/arkmedic/posts/107778812420845822
It's pieces of these that end up in shots, not the air.
https://breggin.com/article-detail/post_detail/Sucharit-Bhakdi-in-Athens-September-24-2022
My daughter sent me this today. Would you please comment on this. I do not know what to think.
Jessica, Zhengli Shi and cohorts proudly answered your question whether an "insert" from HIV was added to the Covid S-protein when they published that THEY DID EXACTLY THAT, in Journal of Virology, Feb. 2008, p. 1899–1907; Vol. 82, No. 4. From the article (emphasis is mine):
“In this study, we investigated the receptor usage of the SL-CoV S by combining a HUMAN IMMUNODEFICIENCY VIRUS-BASED PSEUDOVIRUS SYSTEM with cell lines expressing the ACE2 molecules of human, civet, or horseshoe bat ... In addition to full-length S of SL-CoV and SARS-CoV, a series of S chimeras was constructed by inserting different sequences of the SARS-CoV S into the SL-CoV S backbone ... a minimal insert region (amino acids 310 to 518) was found to be sufficient to convert the SL-CoV S from non-ACE2 binding to human ACE2 binding...”
Of course after working to perfect the human pathogenicity of this monster, Shi later published more of her progress, along with Ralph Baric and 13 other researchers (only one of whom was Chinese) in 2015. This was in Nature Medicine, where again they mentioned using an "HIV-based pseudovirus" and again, the emphasis is mine:
"...DBT cells (Baric laboratory, source unknown) expressing ACE2 orthologs have been previously described for both human and civet; bat Ace2 sequence was based on that from Rhinolophus leschenaulti, and DBT cells expressing bat Ace2 were established as described previously(8). Pseudotyping experiments were similar to those USING AN HIV-BASED PSEUDOVIRUS, prepared as previously described(10), and examined on HeLa cells (Wuhan Institute of Virology) that expressed ACE2 orthologs...."
But don't worry, all this was nothing to be concerned about, because the Nature Medicine article also states: "Synthetic construction of chimeric mutant and full-length SHC014-CoV was approved by the University of North Carolina Institutional Biosafety Committee and the Dual Use Research of Concern committee..." See? Uncle Ralph, said it was okay!
And there you have a YES to your other question, Jessica -- were recombinant techniques used at the Wuhan Institute of Virology to create something incredibly similar to SARS Cov-2, or possibly SARS Cov-2 itself? But did we even need that proof? Cuz... Sars Cov-2 was either engineered, or it naturally come from bats who inserted a Moderna-patented furin cleavage sequence into a virus while in their cave, then flew ~900 miles to Wuhan to spread it, where Chinese and American virologists had been working for years to turn a non-humanly-infectious horseshoe bat coronavirus into a lethal pathogen. And amazingly, those bats did so without infecting anyone along the way...
Here's the doi for the Journal of Virology article: doi:10.1128/JVI.01085-07. And for the Nature Medicine article: doi:10.1038/nm.3985.
Would love your take on those articles, Dr. Rose. And if you would, please use some recombinant techniques yourself, to create duplicates of your phenotype. We could really use a few more admirably relentless, Flamenco-dancing computational biologists who surf, to guide us through the science which is otherwise incomprehensible to us mortals. Thank you, thank you, for having the courage to share your brilliance with the rest of us.
The pandemic has been an odd blessing triggering dynamic alliances among like minded from every discipline & background on emergency footing crowdsourcing research and brainstorming solutions.
Can't wait for Nov 8th.. right up w Simon & Garfunkel in Central Park atm.. flippin adore you all!! tyvm
That was awesome in a strenuous mind stretching manner.
Great detail, and interesting read.
Thank you
bioRxiv is where I first read about the furin cleavage site in a paper by Yuri Deigin (https://medium.com/@yurideigin/lab-made-cov2-genealogy-through-the-lens-of-gain-of-function-research-f96dd7413748)....this was shortly after a young 36-year-old doctor died from thrombocytopenia here in Miami....I was highly suspicious of the global shenanigans before that, but wow, after reading Deighin, I pulled out the tin foil hat...and then I found Team Enigma, then you, and McCullough, La Quinta Columna, and onward...thank you, Jessica. You are on the right side!
I remember well august 2022 with Ana Maria and Karen on Stew Peter Show, testifying that they had found HIV in the injections among fragmental dnas from insects and snakes, graphene and so on. For me I tried to get people´s attention to this, here on Substack, but to no avail until the whole dna fragment scandal was rather recently rolled out. Noone could imagine what has then become common knowledge, thousands of vaxxed deaths and more still coming on within this genocide.
Many years ago (about 1988) I was shooting a game of 8-ball with a med student who had just completed a clinical rotation through urology and had begun his rotation through psychiatry. The med student's urology professor was an old guy, who had done his urology residency at NYC's Bellevue Hospital in the late 1950s. One year a strange epidemic appeared in NYC's Haitian immigrant community, that seemed to be sexually transmitted and resulted in catastrophic failure of the immune system. Nearly all the 30-odd patients admitted with the condition, already had caught at least one secondary infection. All of them had skin lesions resembling Kaposi's Sarcoma, said the urology professor. None of them recovered. Most died within 30 days of hospital admission.
1) Was there a wild-type ancestor of the HIV virus running loose in Haiti in the 1950's?
2) Did it have a natural reservoir in some species found in Haiti?
3) Was HIV a less-lethal variant of the wild-type virus, that left enough living patients around to be studied?
I have zero knowledge beyond that, to contribute. What injected this disturbing news into our game of eight-ball, was my chance comment about interning in organic chemistry at what's now the Karmanos Cancer Institute in Detroit, for Dr Jerome Hurwitz, who instinctively kept closets and fridges full of samples of every hypothetical chemo drug his research group had ever made. Some decades before, one of the Karmanos Institute staff dreamt up a mechanism similar to the yet-undiscovered replication mechanism of retroviruses and postulated there might be cancers that reproduce in that way. After some discussion, they dreamed up the structure of Azidothymidine (AZT), got an NCI grant to study it, made a few dozen grams of it, and tested it in vitro on a number of types of cancer tissue to see if it would block them from growing. Result: Dismal failure. Not one cancer to date, reproduces like a retrovirus does.
Decades later, old Horwitz got a phone call from NIH asking if he had any hints for them on how to make AZT. Upon learning they were thinking it might solve the HIV epidemic, he shared his sample and some old notebook pages. At the time, a public health priority was to persuade at-risk patients to get tested for HIV. The mere possibility of a treatment could incentivize people to learn if they were infected and stop risky behaviors that could spread the infection.
Quite a surprising conversation indeed.
I comprehend about 1% of this.
Regarding lumphocytopenia, what happened to the cytokine storms?
"I just don't know why yet"
I sliced my way to this answer with Occam's razor in one hand and Hanlon's in my other:
what better way to get rid of a germaphobe president? It was also, a dual-pronged attack (a tactic favorited by leftist operatives): Should he win? Two years in, EVERY vaccine injury is blamed on him. OR alternatively, it helps him lose.
EZ PZ.
Hi Jessica, a colleague just wrote this, your the first person I thought of, one b/c this is your knowledge wheel house and you love to investigate. Have you heard about this or did I just miss you writing on it? '“[O]n Wednesday, four months after vaccinations began in young children, Moderna investigators published [a] paper in the New England Journal of Medicine… Deep in the paper’s appendix, Moderna disclosed a case of new-onset Type 1 diabetes in a one-year-old girl that its investigators found was vaccine-related. This is a very serious vaccine-caused side effect that occurred in the trial and as far as I can tell has NEVER been mentioned anywhere else.
On page 62 of the paper’s 68-page appendix, in Table S26, Moderna charts all the side effects reported in the trial, serious or not. The footnotes to the Table S26 contain this note:
“the other SAE [serious adverse event] considered related was new-onset Type 1 diabetes mellitus and diabetic ketoacidosis in a 1-year-old female reported 37 days post dose 2.”
Based on the description in the footnotes, this incident was very dangerous. Diabetic ketoacidosis, or DKA, occurs when the body can no longer rely on its sugars for fuel because of a lack of insulin. Instead, it breaks down fat in an effort to survive, causing acids called ketones to build in the blood. DKA can be fatal if it is not quickly medically managed.
Neither the Centers for Disease Control report nor the Canadian report … mentioned this case; it is not clear why. The Moderna investigators also did not mention the case in the paper itself, only the appendix.
Type 1 diabetes occurs when body’s immune system attacks the pancreas and causes it to stop producing insulin. It is most frequently diagnosed in children and teenagers. It can occur and be diagnosed in infants and toddlers, but such diagnoses are very rare. A 1998 British paper found the annual incidence to be about 1 in 15,000 children.
Case reports from Japan and elsewhere have also linked the mRNA shots to the development of Type 1 diabetes in adults.” Alex Berenson, 10/21/22'