Nerve, brain and blood vessel damage: autoantibodies are the culprit
A new study corroborates attack of self by spike making the Edogawa-McCairn Protocol even more relevant
A new paper was just published in Cell entitled: A causal link between autoantibodies and neurological symptoms in long COVID.1 It was published on May 28, 2026 by a group at Yale and prove that molecular mimicry was always a problem with respect to the spike protein. These published lab confirmations push the biowarfare (as opposed to biodefense) buttons in me, and in light of these findings, acknowledgement by the “authorities” who continue to cover up the truth behind the SARS-2/COVID story is NOT AVOIDABLE. Early acknowledgement of numerous published findings from years ago could have been followed by immediate treatment (Edogawa-McCairn Protocol) to prevent long-term damage in millions.
People have been treated like cattle to be slaughtered with impunity - like casualties of some private bio war - and those responsible for continuing to allow this barbaric behaviour of human beings are as responsible as those who started it all.
Here’s what the authors found.
“Long COVID”2 can involve antibody generation directed at self (we call these autoantibodies/autoimmunity) that specifically target nerves, the brain, and blood vessels. After testing patient antibodies against thousands of human proteins for autoantibody effects, the most striking observation they made was that these antibodies are extra aggressive against a specific human protein. This specific protein called MED20 (Mediator Complex Subunit 20) is really important because it’s part of a large mediator complex inside our cells that helps turn genes on and off. Basically, it acts like a control switch for how cells read their DNA and make proteins. Kind of important, no? People with mutations in the MED20 gene are associated with basal ganglia degeneration and brain atrophy.34
Since the antibodies from the people in the study showed stronger activity against MED20, this would inevitably cause the immune system to attack healthy tissues more than normal. Furthermore, when they passed the human autoantibodies into mice, the mice developed pain and fatigue-like symptoms - similar to what humans report.
Not only that, but the worse the mouse symptoms were, the worse the patient’s pain. The level of pain-related behavior in the mice closely matched how much chronic pain the original patients were experiencing so it’s not a stretch to conclude that the autoantibodies are directly contributing to these symptoms.
It frustrates me to no end that I have written countless articles on molecular mimicry due to homologous peptides known to be present in the spike protein, but apparently to deaf ears. I am not alone in my conclusions at all. Open-source bioinformatics tools have existed for a long time and are extremely easy to use. Anyone designing a gene-based pro-drug meant to be forced into billions of people should have used ALL OF THEM to rule out potential amyloidogenic proteins and proteins with homology to human proteins that might lead to autoimmunity.
This paper entitled Molecular Mimicry Map (3M) of SARS-CoV-2: Prediction of potentially immunopathogenic SARS-CoV-2 epitopes via a novel immunoinformatic approach, has been sitting (and rotting) on the bioRxiv preprint server since November 12, 2020. There’s simply no excuse for any of this to be happening. We have the resources, the minds and the technology to not only heal people, but to prevent them from being damaged in the first place at the hands of “public health”.
Here are just a few of the articles I have written over the years for posterity.
Autoimmunity and tolerance
During my Immunology degree program, we used many textbooks to enhance our understanding of all things immunity. Kuby, Janeway, Parham, Wood - each one with a bunch of pros and cons. I liked Kuby. Janeway was always the go-to. The latter written by Peter Wood called “Understanding immunology”
A short compendium of evidence to date...
I have become interested lately in more than just reporting on adverse event data and what’s going on in the peer-reviewed literature. I seem to be approaching a larger loop in the life spiral where I feel it’s time to summarize. So I attempted to do so in a meeting of doctors and lawyers last night. Here are some of the slides I presented. Feel free to…
Molecular mimicry of SARS-nCoV-2 spike to human proteins including thrombopoietin and TLR-8
Please refer to the paper entitled: “Potential Autoimmunity Resulting from Molecular Mimicry between SARS-CoV-2 Spike and Human Proteins” published June 28, 2022 in Viruses.
Molecular mimicry shown between Multiple Sclerosis-associated proteins and SARS-2 nucleocapsid
A paper is hot off the presses, published in Scientific Reports 2 days ago (Jan 8, 2022) entitled: “Sequence similarity between SARS-CoV-2 nucleocapsid and multiple sclerosis-associated proteins provides insight into viral neuropathogenesis following infection
Interstitial lung disease, MDA5-positive dermatomyositis and ties to SARS-CoV-2 spike protein
Someone in the Twitter world tagged me in a post yesterday with an excellent question. He wanted to know if a strange and rare disease that ‘nobody has ever survived’ called MDA5-positive dermatomyositis (anti-MDA5 dermatomyositis), was possibly the result of a ‘wrong immune response’ instigated by the injections. He was asking in the context of a young…
VAERS reports contradict claim of no AEs in frameshifting context
“All of the harms from the COVID-19 injectable products were predictable, and preventable.”
This new study makes the Edogawa-McCairn Protocol even more relevant, and it is important to make it available to anyone who wants to try it, especially those who are suffering badly. Although it is an experimental protocol (for now), it has demonstrated roaring success in 17 people to date, including for a dear friend of mine Charles Rixey (a “vaccine” -injured U.S. Marine), and many more are lining up for this treatment.
It is being done at Edogawa Hospital in Japan and it involves Double Filtration Plasmapheresis (DFPP) to filter out harmful autoantibodies, spike protein, and amyloid-like microclots from the blood. It is followed by infusions of dental pulp stem cell-derived growth factors to reduce inflammation and promote recovery in patients with severe injury.
Dr. Mary Talley Bowden, a board-certified doctor in Otolaryngology and Sleep Medicine, is trying to bring this protocol to the U.S. so that people who can’t afford a ticket to Japan may have access to this treatment. IRBs pending. More hoops to jump through while people suffer - all in the name of “public health”.
Early acknowledgement and treatment could have prevented long-term damage in millions and I don’t know about you guys, but I don’t need some strange idea of an authority figure to give me permission to do what I want to do with my body or to heal myself as I see fit, especially considering that all of these authority-figure impositions and rules and regulations seem to be very counter-indicative to actual health and longevity.
I hope that many more human beings now realize that the only true authority is the self.
Stay tuned.
de Sá KSG, Silva J, Bayarri-Olmos R, et al. A causal link between autoantibodies and neurological symptoms in long COVID. Cell. 2026;189(11):3214-3235.e37. doi:10.1016/j.cell.2026.05.009
The reader must know that I have never believed in the idea of “long COVID”. I believe it is nothing more than sustained activation of latent viruses and autoimmune reactions from spike protein - which is precisely what this paper demonstrates.
Tang, Wen-Shuai; et al. (July 2021). “The Mediator subunit MED20 organizes the early adipogenic complex to promote development of adipose tissues and diet-induced obesity”. Cell Reports. 36 (1) 109314. doi:10.1016/j.celrep.2021.109314
Vodopiutz, Julia; Schmook, Maria T.; Konstantopoulou, Vassiliki; Plecko, Barbara; Greber-Platzer, Susanne; Creus, Marc; Seidl, Rainer; Janecke, Andreas R. (January 2015). “MED20 mutation associated with infantile basal ganglia degeneration and brain atrophy”. European Journal of Pediatrics. 174 (1): 113–118. doi:10.1007/s00431-014-2463-7
Is it safe to assume that somewhere at Pfizer, Moderna and the DofD there must be piles of animal research data that would confirm that all these negative outcomes were known about BEFORE the vaccines were hoisted on the general public? Why aren't we seeing those kinds of basic questions put forward by anyone in Washington or the media? Why has no one ever been confronted anyone about the bait-and-switch with Process 1 versus Process 2 refinement? What does their pre-human trial data reveal about the difference in negative outcome between the two processes? I know we'll never get these kinds of answers, I just hope to continue to avoid any and all mRNA injectables these monsters continue to push on us
Brilliant article. Did Not Know that you Are also Masters in autoimmunity. But as I learned with my microbiome Mikroskope and in 5000 HLA-B27 transgenic autoimmune ancylosing spondylitis mixe Models microbes Are always a useful cause of any autoimmunity as the immune System is Not stupid. We found Spike Gene Transfected aspergillus and candida in vaccinated and non vaccinated people, even cancer from Spike fungi in non vaccinated. So ist much more simple than autoimmunity. Irun Cohen was Not Right. Real autoimmunity would mean that our immune System is stupid. It is not. You always find a causative microbe expanding too much in the Body. Because of toxins.